In cyanotic heart disease why increased pulmonary blood flow

In cyanotic heart disease why increased pulmonary blood flow (Mixing lesion) is also a type included?

Because already in acyanotic heart disease it is included due to left to right shunts.

Given in module 39 of paediatrics in the app and time at 2:02 minutes.

Cyanotic congenital heart diseases are characterized by a decreased amount of oxygenated blood being delivered to the body, resulting in cyanosis (a bluish discoloration of the skin and mucous membranes). These conditions are often associated with right-to-left shunting of blood, either at the level of the heart or within the great vessels.

However, it’s important to clarify that increased pulmonary blood flow (often referred to as a “mixing lesion”) is not a specific type of cyanotic congenital heart disease. Instead, increased pulmonary blood flow is a hemodynamic finding that can be present in both cyanotic and acyanotic congenital heart diseases.

In the context of congenital heart diseases, “increased pulmonary blood flow” generally refers to a condition where there is an abnormal communication or shunting of blood from the left side of the heart (systemic circulation) to the right side (pulmonary circulation), thereby increasing the blood flow to the lungs. This can occur due to various types of heart defects, including septal defects (like atrial septal defect or ventricular septal defect), patent ductus arteriosus (PDA), and atrioventricular septal defect (AVSD).

In cyanotic heart diseases, the term “increased pulmonary blood flow” is often used to describe the presence of additional shunting of blood from the right side to the left side of the heart (right-to-left shunt) or intrapulmonary shunting, which further contributes to decreased systemic oxygenation and exacerbates cyanosis.

To summarize, “increased pulmonary blood flow” is a hemodynamic finding that can be seen in both cyanotic and acyanotic congenital heart diseases, but it is not a specific type of cyanotic heart disease. It refers to abnormal shunting of blood that results in an increased blood flow to the lungs, often associated with specific types of congenital heart defects.